Lack of IFNγ signaling attenuates spread of influenza A virus in vivo and leads to reduced pathogenesis-Reference-Cited by-同舟云学术

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Lack of IFNγ signaling attenuates spread of influenza A virus in vivo and leads to reduced pathogenesis

Published:2019-01 Issue: Volume:526 Page:155-164
ISSN:0042-6822
Container-title:Virology
language:en
Short-container-title:Virology

Author:

Nicol Marlynne Q.,Campbell Gillian M.,Shaw Darren J.,Dransfield Ian,Ligertwood Yvonne,Beard Philippa M.,Nash Anthony A.,Dutia Bernadette M.

Funder

The Roslin Institute

Interdisciplinary Centre for Human and Avian Influenza Research

ICHAIR-BBSRC Studentship

Publisher

Elsevier BV

Subject

Virology

Reference36 articles.

1. Interleukins, from 1 to 37, and interferon-gamma: receptors, functions, and roles in diseases;Akdis;J. Allergy Clin. Immunol.,2011

2. In vivo blockade of gamma interferon affects the influenza virus-induced humoral and the local cellular immune response in lung tissue;Baumgarth;J. Virol.,1996

3. Protective role of gamma interferon during the recall response to influenza virus;Bot;J. Virol.,1998

4. IFN-gamma increases susceptibility to influenza A infection through suppression of group II innate lymphoid cells;Califano;Mucosal Immunol.,2017

5. Susceptibility of bone marrow-derived macrophages to influenza virus infection is dependent on macrophage phenotype;Campbell;J. Gen. Virol.,2015

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