Herpes simplex virus type 2-mediated disease is reduced in mice lacking RNase L
Author:
Publisher
Elsevier BV
Subject
Virology
Reference47 articles.
1. The murine double-stranded RNA-dependent protein kinase PKR and the murine 2′,5′-oligoadenylate synthetase-dependent RNase L are required for IFNβ-mediated resistance against herpes simplex virus type 1 in primary trigeminal ganglion culture;Al-khatib;Virology,2003
2. Distinctive roles for 2′,5′-oligoadenylate synthetases and double-stranded RNA-dependent protein kinase R in the in vivo antiviral effect of an adenoviral vector expressing murine IFN-beta;Al-khatib;J. Immunol.,2004
3. Dichotomy between survival and lytic gene expression in RNase L- and PKR-deficient mice transduced with an adenoviral vector expressing murine IFN-beta following ocular HSV-1 infection;Al-khatib;Exp. Eye Res.,2005
4. Critical role for the oligoadenylate synthetase/RNase L pathway in response to IFN-β during acute ocular herpes simplex virus type 1 infection;Austin;J. Immunol.,2005
5. Interferon-β suppresses herpes simplex virus type 1 replication in trigeminal ganglion cells through an RNase L-dependent pathway;Carr;J. Neuroimmunol.,2003
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