PLAC8 promotes EV71 infected inflammatory lesion by disturbing Th-cell-related cytokines release in neonatal mouse
Author:
Yang XiaoxiaORCID,
Shui Xiaochuan,
Dai Xiaoqing,
Hao Shiyong,
Ke Feng,
Zhu Liru,
Chen Xuejiao
Reference20 articles.
1. The current status of the disease caused by enterovirus 71 infections: epidemiology, pathogenesis, molecular epidemiology, and vaccine development;Chang;Int. J. Environ. Res. Publ. Health,2016
2. PLAC8, a new marker for human interstitial extravillous trophoblast cells, promotes their invasion and migration;Chang;Development,2018
3. Enhanced type 2 and diminished type 1 cytokines in neonatal tolerance;Chen;Transplantation,1995
4. Sustained high levels of interleukin-6 contribute to the pathogenesis of enterovirus 71 in a neonate mouse model;Khong;J. Virol.,2011
5. Neonatal innate TLR-mediated responses are distinct from those of adults;Kollmann;J. Immunol.,2009
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