Short chain fatty acids potently induce latent HIV-1 in T-cells by activating P-TEFb and multiple histone modifications-Reference-Cited by-同舟云学术

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Short chain fatty acids potently induce latent HIV-1 in T-cells by activating P-TEFb and multiple histone modifications

Published:2015-01 Issue: Volume:474 Page:65-81
ISSN:0042-6822
Container-title:Virology
language:en
Short-container-title:Virology

Author:

Das Biswajit,Dobrowolski Curtis,Shahir Abdel-Malek,Feng Zhimin,Yu Xiaolan,Sha Jinfeng,Bissada Nabil F.,Weinberg Aaron,Karn Jonathan,Ye Fengchun^ORCID

Funder

National Institute of Dental & Craniofacial Research of the National Institutes of Health to Fengchun Ye and Jonathan Karn

CWRU/UH Center for AIDS Research (CFAR) to Fengchun Ye

Jonathan Karn

Publisher

Elsevier BV

Subject

Virology

Reference101 articles.

1. Butyrate and propionate induced activated or non-activated neutrophil apoptosis via HDAC inhibitor activity but without activating GPR-41/GPR-43 pathways;Aoyama;Nutrition,2010

2. Influence of coinfecting pathogens on HIV expression: evidence for a role of Toll-like receptors;Bafica;J. Immunol,2004

3. Histone deacetylase inhibitors (HDACis) that release the positive transcription elongation factor b (P-TEFb) from its inhibitory complex also activate HIV transcription;Bartholomeeusen;J. Biol. Chem.,2013

4. The Suv39H1 methyltransferase inhibitor chaetocin causes induction of integrated HIV-1 without producing a T cell response;Bernhard;FEBS Lett,2011

5. Recruitment of a protein complex containing Tat and cyclin T1 to TAR governs the species specificity of HIV-1 Tat;Bieniasz;EMBO J.,1998

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