Functional F11L and K1L genes in modified vaccinia virus Ankara restore virus-induced cell motility but not growth in human and murine cells
Author:
Funder
European Commission
Publisher
Elsevier BV
Subject
Virology
Reference37 articles.
1. The complete genomic sequence of the modified vaccinia Ankara strain: comparison with other orthopoxviruses;Antoine;Virology,1998
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3. Viral host-range factor C7 or K1 is essential for modified vaccinia virus Ankara late gene expression in human and murine cells, irrespective of their capacity to inhibit protein kinase R-mediated phosphorylation of eukaryotic translation initiation factor 2
4. Generation of recombinant fowlpox virus using the non-essential F11L orthologue as insertion site and a rapid transient selection strategy;Boulanger;J. Virol. Meth.,2002
5. Vaccinia virus K1L protein mediates host-range function in RK-13 cells via ankyrin repeat and may interact with a cellular GTPase-activating protein;Bradley;Virus Res.,2005
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2. SPI-1 is a missing host-range factor required for replication of the attenuated modified vaccinia Ankara (MVA) vaccine vector in human cells;PLOS Pathogens;2019-05-30
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5. Vaccinia virus F5 is required for normal plaque morphology in multiple cell lines but not replication in culture or virulence in mice;Virology;2014-05
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