The HCMV US28 vGPCR induces potent Gαq/PLC-β signaling in monocytes leading to increased adhesion to endothelial cells
Author:
Funder
National Institutes of Health
Publisher
Elsevier BV
Subject
Virology
Reference70 articles.
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1. Delivery of US28 by incoming HCMV particles rapidly attenuates Akt activity to suppress HCMV lytic replication in monocytes;Science Signaling;2024-08-27
2. CMV-encoded GPCRs in infection, disease, and pathogenesis;Advances in Virus Research;2024
3. Virion-associated US28 rapidly modulates Akt activity to suppress HCMV lytic replication in monocytes;2023-09-05
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5. Cytomegalovirus US28 regulates cellular EphA2 to maintain viral latency;Science Advances;2022-10-28
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