Characterization of de novo lytic infection of dermal lymphatic microvascular endothelial cells by Kaposi's sarcoma-associated herpesvirus
Author:
Funder
American Cancer Society Research Scholar
NIH
UF Health Cancer Center
UF Research Opportunity Seed Fund
NIH Training
Publisher
Elsevier BV
Subject
Virology
Reference35 articles.
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3. Molecular and cellular mechanisms of KSHV oncogenesis of Kaposi's sarcoma associated with HIV/AIDS;Cavallin;PLoS Pathog.,2014
4. Gammaherpesvirus and lymphoproliferative disorders in immunocompromised patients;Cesarman;Cancer Lett.,2011
5. A unique herpesviral transcriptional program in KSHV-infected lymphatic endothelial cells leads to mTORC1 activation and rapamycin sensitivity;Chang;Cell Host Microbe,2013
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1. A specific and portable gene expression program underlies antigen archiving by lymphatic endothelial cells;2024-04-02
2. Kaposi’s sarcoma herpesvirus latency-associated nuclear antigen broadly regulates viral gene expression and is essential for lytic infection;PLOS Pathogens;2024-01-17
3. An Essential Role of c-Fos in Notch1-mediated Promotion of Proliferation of KSHV-Infected SH-SY5Y Cells;Current Molecular Pharmacology;2024-01-11
4. Hypoxia and hypoxia‐inducible factors in Kaposi sarcoma‐associated herpesvirus infection and disease pathogenesis;Journal of Medical Virology;2023-09
5. Kaposi sarcoma-associated herpesvirus latency-associated nuclear antigen: more than a key mediator of viral persistence;Current Opinion in Virology;2023-08
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