p53 suppresses the inflammatory response following respiratory syncytial virus infection by inhibiting TLR2
Author:
Funder
National Natural Science Foundation of China
Fundamental Research Funds for the Key Research Program of Chongqing Science and Technology Commission
Publisher
Elsevier BV
Reference46 articles.
1. Interaction of ectodomain of respiratory syncytial virus G protein with TLR2/TLR6 heterodimer: an in vitro and in silico approach to decipher the role of RSV G protein in pro-inflammatory response against the virus;Alshaghdali;Curr. Pharmaceut. Des.,2021
2. Respiratory syncytial virus matrix protein induces lung epithelial cell cycle arrest through a p53 dependent pathway;Bian;PLoS One,2012
3. P53 vs NF-kappaB: the role of nuclear factor-kappa B in the regulation of p53 activity and vice versa;Carra;Cell. Mol. Life Sci.,2020
4. Potential role for p53 in the permissive life cycle of human cytomegalovirus;Casavant;J. Virol.,2006
5. The fusion protein of respiratory syncytial virus triggers p53-dependent apoptosis;Eckardt-Michel;J. Virol.,2008
Cited by 2 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Immunomodulatory markers and therapies for the management of infant respiratory syncytial virus infection;Expert Review of Anti-infective Therapy;2024-09-13
2. Inflammatory response to SARS-CoV 2 and other respiratory viruses;Expert Review of Anti-infective Therapy;2024-09-11
1.学者识别学者识别
2.学术分析学术分析
3.人才评估人才评估
"同舟云学术"是以全球学者为主线，采集、加工和组织学术论文而形成的新型学术文献查询和分析系统，可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容，当前同舟云学术共收录了国内外主流学术期刊6万余种，收集的期刊论文及会议论文总量共计约1.5亿篇，并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询！咨询电话：010-8811{复制后删除}0370
www.globalauthorid.com
TOP
Copyright © 2019-2024 北京同舟云网络信息技术有限公司 京公网安备11010802033243号 京ICP备18003416号-3