A drug-selected Plasmodium falciparum lacking the need for conventional electron transport-Reference-Cited by-同舟云学术

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A drug-selected Plasmodium falciparum lacking the need for conventional electron transport

Published:2008-05 Issue:1 Volume:159 Page:64-68
ISSN:0166-6851
Container-title:Molecular and Biochemical Parasitology
language:en
Short-container-title:Molecular and Biochemical Parasitology

Author:

Smilkstein Martin J.,Forquer Isaac,Kanazawa Atsuko,Kelly Jane Xu,Winter Rolf W.,Hinrichs David J.,Kramer David M.,Riscoe Michael K.

Publisher

Elsevier BV

Subject

Molecular Biology,Parasitology

Reference27 articles.

1. Uncovering the molecular mode of action of the antimalarial drug atovaquone using a bacterial system;Mather;J Biol Chem,2005

2. Mitochondrial drug targets in apicomplexan parasites;Mather;Curr Drug Targets,2007

3. Metabolic maps and functions of the Plasmodium mitochondrion;van Dooren;FEMS Microbiol Rev,2006

4. Site of action of the antimalarial hydroxynaphthoquinone, 2-[trans-4-(4′-chlorophenyl) cyclohexyl]-3-hydroxy-1,4-naphthoquinone (566C80);Fry;Biochem Pharmacol,1992

5. Atovaquone, a broad spectrum antiparasitic drug, collapses mitochondrial membrane potential in a malarial parasite;Srivastava;J Biol Chem,1997

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3. Reaction hijacking of tyrosine tRNA synthetase as a new whole-of-life-cycle antimalarial strategy;Science;2022-06-03

4. Robenidine Analogues Are Potent Antimalarials in Drug-Resistant Plasmodium falciparum;ACS Infectious Diseases;2021-03-16

5. Atypical Molecular Basis for Drug Resistance to Mitochondrial Function Inhibitors in Plasmodium falciparum;Antimicrobial Agents and Chemotherapy;2021-02-17

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