Effects of intravenous fluorocarbons during and without oxygen enhancement on acute myocardial ischemic injury assessed by measurement of intramyocardial gas tensions-Reference-Cited by-同舟云学术

Effects of intravenous fluorocarbons during and without oxygen enhancement on acute myocardial ischemic injury assessed by measurement of intramyocardial gas tensions

Published:1982-06 Issue:6 Volume:103 Page:986-995
ISSN:0002-8703
Container-title:American Heart Journal
language:en
Short-container-title:American Heart Journal

Author:

Rude Robert E.,Glogar Dietmar,Khuri Shukri F.,Kloner Robert A.,Karaffa Stephanie,Muller James E.,Clark Leland C.,Braunwald Eugene

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine

Reference39 articles.

1. Survival of mammals breathing organic liquids equilibrated with oxygen at atmospheric pressure;Clark;Science,1966

2. Organ perfusion with fluorocarbon fluid (abstr);Gollan;Physiologist,1966

3. Erythrocyte substitute for perfusion of brain;Sloviter;Nature,1967

4. Survival of rats having red cells totally replaced with emulsified fluorocarbons (abstr);Geyer,1968

5. Perfusion of whole animals with perfluorinated liquid emulsions using the Clark bubble-defoam heart-lung machine;Clark,1970

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1. Perfluorocarbon Induced Alterations in Pulmonary Mechanics;Artificial Cells, Blood Substitutes, and Biotechnology;1998-01

2. Influence of Intravenous Perfluorocarbon Administration on the Dynamic Behavior of Lung Surfactant;Artificial Cells, Blood Substitutes, and Biotechnology;1998-01

3. Effects of perfluorocarbon exchange transfusion on reducing myocardial infarct size in a primate model of ischemia-reperfusion injury: A prospective, randomized study;Surgery;1995-06

4. Effects of A 100% Perfluorooctylbromide Emulsion on Ischemia/Reperfusion Injury Following Cardioplegia;Artificial Cells, Blood Substitutes, and Biotechnology;1995-01

5. Hyperoxic reperfusion is required to reduce infarct size after intravenous therapy with perfluorochemical (Fluosol-DA 20%) or its detergent component (poloxamer 188) in a poorly collateralized animal model;Journal of the American College of Cardiology;1994-10