Catechin and epicatechin reduce mitochondrial dysfunction and oxidative stress induced by amiodarone in human lung fibroblasts

Author:

Silva Santos Luciana Fernandes,Stolfo Adriana,Calloni Caroline,Salvador Mirian

Publisher

Wiley

Subject

Cardiology and Cardiovascular Medicine

Reference36 articles.

1. Cardiac arrhythmias: diagnosis, symptoms, and treatments;Fu;Cell Biochem Biophys,2015

2. Detection of amiodarone-induced pulmonary toxicity in supine and prone positions;Oyama;Circ J,2005

3. WHO - NHS Centre for the evaluation of effectiveness of health care (CeVEAS). 17th Expert Committee on the Selection and Use of Essential Medicines. Proposal for the Inclusion of Amiodarone as an Anti-arrhythmic and/or for the Treatment of Chronic Heart Failure in the Who Model List of Essential Medicines. Geneva. Available online in: 〈http://www.who.int/selection_medicines/committees/expert/17/application/amiodarone_inclusion.pdf〉. 2009, [accessed on 5.7.2012]

4. Effects of Amiodarone and N- desethylamiodarone on Cardiac Voltage-Gated Sodium Channels;Ghovanloo;Front Pharmacol,2016

5. The role of CYP 3A4 and 1A1 in amiodarone-induced hepatocellular toxicity;Wu;Toxicol Lett,2016

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