Whole-heart computational modelling provides further mechanistic insights into ST-elevation in Brugada syndrome

Author:

Wülfers Eike M.,Moss Robin,Lehrmann Heiko,Arentz Thomas,Westermann Dirk,Seemann Gunnar,Odening Katja E.,Steinfurt Johannes

Funder

University of Freiburg Faculty of Medicine

Publisher

Elsevier BV

Reference49 articles.

1. Brugada syndrome: clinical, genetic, molecular, cellular, and ionic aspects;Antzelevitch;Curr. Probl. Cardiol.,2016

2. Genome-wide association analyses identify new Brugada syndrome risk loci and highlight a new mechanism of sodium channel regulation in disease susceptibility;Barc;Nat. Genet.,2022

3. A novel rule-based algorithm for assigning myocardial fiber orientation to computational heart models;Bayer;Ann. Biomed. Eng.,2012

4. Brugada syndrome and reduced right ventricular outflow tract conduction reserve: a final common pathway?;Behr;Eur. Heart J.,2021

5. Diffuse fibrosis and repolarization disorders explain ventricular arrhythmias in Brugada syndrome: a computational study;Biasi;Sci. Rep.,2022

Cited by 2 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献

1. Electrophysiological patterns and structural substrates of Brugada syndrome: Critical appraisal and computational analyses;Journal of Cardiovascular Electrophysiology;2024-06-20

2. Digital Twins of Biological Systems: A Narrative Review;IEEE Open Journal of Engineering in Medicine and Biology;2024

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