Diisopropylfluorophosphate-induced depression of segmental monosynaptic transmission in neonatal rat spinal cord is also mediated by increased axonal activity
Author:
Publisher
Elsevier BV
Subject
Toxicology,General Medicine
Reference15 articles.
1. Segmental synaptic depression caused by diisopropylphosphorofluoridate and sarin is reversed by thyrotropin-releasing hormone in the neonatal rat spinal cord;DasGupta;Toxicol. Appl. Pharmacol.,1988
2. Interaction of reversible and irreversible cholinesterase inhibitors on the monosynaptic reflex in neonatal rats;DasGupta;Toxicol. Appl. Pharmacol.,1989
3. Effect of organophosphates and carbamates on the monosynaptic reflex recorded from neonatal rat spinal cord;DasGupta,1993
4. Biphasic action of sarin on monosynaptic reflex in the neonatal rat spinal cord in vitro;Warnick;Arch. Toxicol.,1993
5. Central effects of centripetal impulses in axons of spinal ventral roots;Renshaw;J. Neurophysiol.,1946
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1. Ptychodiscus brevis toxin enhances the frequency-dependent depression of the monosynaptic reflex in neonatal rat spinal cord in vitro;Toxicology Letters;2003-12
2. Deprenyl blocks the aglycemia-induced depression of the synaptic transmission but not the ischemia-induced depression in neonatal rat spinal cord in vitro;Neuroscience Research;2003-09
3. Involvement of N-methyl-D-aspartate receptors for the Ptychodiscus brevis toxin-induced depression of monosynaptic and polysynaptic reflexes in neonatal rat spinal cord in vitro;Neuroscience;2002-12
4. Relative potency of synthetic analogs of Ptychodiscus brevis toxin in depressing synaptic transmission evoked in neonatal rat spinal cord in vitro;Toxicology Letters;2002-03
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