Binge-like ethanol exposure during the brain growth spurt disrupts the function of retrosplenial cortex-projecting anterior thalamic neurons in adolescent mice-Reference-Cited by-同舟云学术

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Binge-like ethanol exposure during the brain growth spurt disrupts the function of retrosplenial cortex-projecting anterior thalamic neurons in adolescent mice

Published:2023-12 Issue: Volume:241 Page:109738
ISSN:0028-3908
Container-title:Neuropharmacology
language:en
Short-container-title:Neuropharmacology

Author:

Bird Clark W.^ORCID,Mayfield Stefanie S.,Lopez Katalina M.,Dunn Brooke R.^ORCID,Feng Angela,Roberts Bryce T.^ORCID,Almeida Roberto N.,Chavez Glenna J.^ORCID,Valenzuela C. Fernando

Publisher

Elsevier BV

Subject

Cellular and Molecular Neuroscience,Pharmacology

Reference64 articles.

1. Why do lesions in the rodent anterior thalamic nuclei cause such severe spatial deficits?;Aggleton;Neurosci. Biobehav. Rev.,2015

2. Excipient exposure in very low birth weight preterm neonates;Akinmboni;J. Perinatol.,2018

3. Third trimester-equivalent ethanol exposure increases anxiety-like behavior and glutamatergic transmission in the basolateral amygdala;Baculis;Pharmacol. Biochem. Behav.,2015

4. Neonatal ethanol exposure triggers apoptosis in the murine retrosplenial cortex: role of inhibition of NMDA receptor-driven action potential firing;Bird;Neuropharmacology,2020

5. Enhancement of parvalbumin interneuron-mediated neurotransmission in the retrosplenial cortex of adolescent mice following third trimester-equivalent ethanol exposure;Bird;Sci. Rep.,2021

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