Polo-like kinase 2 phosphorylation of amyloid precursor protein regulates activity-dependent amyloidogenic processing-Reference-Cited by-同舟云学术

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Polo-like kinase 2 phosphorylation of amyloid precursor protein regulates activity-dependent amyloidogenic processing

Published:2017-05 Issue: Volume:117 Page:387-400
ISSN:0028-3908
Container-title:Neuropharmacology
language:en
Short-container-title:Neuropharmacology

Author:

Lee Yeunkum^ORCID,Lee Ji Soo,Lee Kea Joo,Turner R. Scott,Hoe Hyang-Sook,Pak Daniel T.S.

Funder

Georgetown Memory Disorders Program, Georgetown Partners in Research

National Institute of Health

NIH/NIC CCSG

Publisher

Elsevier BV

Subject

Cellular and Molecular Neuroscience,Pharmacology

Reference48 articles.

1. Tyr(682) in the intracellular domain of APP regulates amyloidogenic APP processing in vivo;Barbagallo;PLoS One,2010

2. Culturing pyramidal neurons from the early postnatal mouse hippocampus and cortex;Beaudoin;Nat. Protoc.,2012

3. Neuronal activity regulates the regional vulnerability to amyloid-β deposition;Bero;Nat. Neurosci.,2011

4. Molecular, structural, and functional characterization of Alzheimer's disease: evidence for a relationship between default activity, amyloid, and memory;Buckner;J. Neurosci.,2005

5. Clusters of hyperactive neurons near amyloid plaques in a mouse model of Alzheimer's disease;Busche;Science,2008

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