Oxaliplatin-induced neurotoxicity is mediated through gap junction channels and hemichannels and can be prevented by octanol-Reference-Cited by-同舟云学术

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Oxaliplatin-induced neurotoxicity is mediated through gap junction channels and hemichannels and can be prevented by octanol

Published:2015-10 Issue: Volume:97 Page:289-305
ISSN:0028-3908
Container-title:Neuropharmacology
language:en
Short-container-title:Neuropharmacology

Author:

Kagiava Alexia,Theophilidis George,Sargiannidou Irene,Kyriacou Kyriacos,Kleopa Kleopas A.

Funder

Cyprus Telethon

Muscular Dystrophy Association

European Leukodystrophy Association

Publisher

Elsevier BV

Subject

Cellular and Molecular Neuroscience,Pharmacology

Reference101 articles.

1. The chemotherapeutic oxaliplatin alters voltage-gated Na(+) channel kinetics on rat sensory neurons;Adelsberger;Eur. J. Pharmacol.,2000

2. Connexin29 is uniquely distributed within myelinating glial cells of the central and peripheral nervous systems;Altevogt;J. Neurosci.,2002

3. Oxaliplatin, fluorouracil, and leucovorin as adjuvant treatment for colon cancer;Andre;N. Engl. J. Med.,2004

4. Structural abnormalities and deficient maintenance of peripheral nerve myelin in mice lacking the gap junction protein connexin32;Anzini;J. Neurosci.,1997

5. Effect of synthetic eel calcitonin, elcatonin, on cold and mechanical allodynia induced by oxaliplatin and paclitaxel in rats;Aoki;Eur. J. Pharmacol.,2012

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