Contribution of dopamine to mitochondrial complex I inhibition and dopaminergic deficits caused by methylenedioxymethamphetamine in mice-Reference-Cited by-同舟云学术

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Contribution of dopamine to mitochondrial complex I inhibition and dopaminergic deficits caused by methylenedioxymethamphetamine in mice

Published:2015-06 Issue: Volume:93 Page:124-133
ISSN:0028-3908
Container-title:Neuropharmacology
language:en
Short-container-title:Neuropharmacology

Author:

Barros-Miñones L.,Goñi-Allo B.,Suquia V.,Beitia G.,Aguirre N.,Puerta E.

Funder

Ministerio de Ciencia e Innovación

Publisher

Elsevier BV

Subject

Cellular and Molecular Neuroscience,Pharmacology

Reference59 articles.

1. MDMA (‘ecstasy’) enhances 5-HT1A receptor density and 8-OH-DPAT-induced hypothermia: blockade by drugs preventing 5-hydroxytryptamine depletion;Aguirre;Eur. J. Pharmacol.,1998

2. Monoamine oxidase-B mediates ecstasy-induced neurotoxic effects to adolescent rat brain mitochondria;Alves;J. Neurosci. Off. J. Soc. Neurosci.,2007

3. Pro-oxidant effects of ecstasy and its metabolites in mouse brain synaptosomes;Barbosa;Br. J. Pharmacol.,2012

4. Dopamine toxicity involves mitochondrial complex I inhibition: implications to dopamine-related neuropsychiatric disorders;Ben-Shachar;Biochem. Pharmacol.,2004

5. Dopamine oxidation alters mitochondrial respiration and induces permeability transition in brain mitochondria: implications for Parkinson's disease;Berman;J. Neurochem.,1999

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