Development of chronic hepatic porphyria (porphyria cutanea tarda) with inherited uroporphyrinogen decarboxylase deficiency under exposure to dioxin

Author:

Doss M.,Sauer H.,Von Tiepermann R.,Colombi A.M.

Publisher

Elsevier BV

Subject

Biochemistry

Reference35 articles.

1. Environmental and drug factors in hepatic porphyria;Bickers;Acta derm. vener. Stockh.,1982

2. Porphyrogenic effect of chronic treatment with 2,3,7,8-tetrachlorodibenzo-p-dioxin in female rats. Dose-effect relationship following urinary excretion of porphyrins;Cantoni;Toxic, appl. Pharmac.,1981

3. Coproporphyrinuria and chronic hepatic porphyria type A found in people from Seveso (Italy) exposed to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD);Centen,1979

4. Subjective symptomatology prevalence as an additional criterion to define riskgroups exposed to TCDD in the Seveso area, Italy;Colombi,1979

5. Analytical and preparative thin-layer chromatography of porphyrin methyl esters;Doss;Z. klin. Chem. klin. Biochem.,1970

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1. Skin diseases associated with Agent Orange and other organochlorine exposures;Journal of the American Academy of Dermatology;2016-01

2. Non-classical congenital adrenal hyperplasia in association with porphyria cutanea tarda: co-incidence or trigger?;Annals of Clinical Biochemistry: International Journal of Laboratory Medicine;2012-07-10

3. Adverse Health Effects in Humans Exposed to 2,3,7,8-Tetrachlorodibenzo-p-Dioxin (TCDD);Reviews on Environmental Health;2006-01

4. The Porphyrias;Zakim and Boyer's Hepatology;2006

5. Approaches to Treatment and Prevention of Human Porphyrias;The Porphyrin Handbook;2003

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