Overproduction of a kinetic subclass of VLDL-apoB, and direct catabolism of VLDL-apoB in human endogenous hypertriglyceridemia: an analytical model solution of tracer data.
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Endocrinology,Biochemistry
Reference38 articles.
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3. The metabolism of apolipoprotein B in subjects with hypertriglyceridemia and polydisperse LDL;Fisher;J. Lipid Res.,1980
4. Kinetic bases of the primary hyperlipidaemias: studies of apolipoprotein B turnover in genetically defined subjects;Janus;Eur. J. Clin. Invest.,1980
5. Very low density lipoprotein overproduction in genetic forms of hypertriglyceridaemia;Chait;Eur. J. Clin. Invest.,1980
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1. Kinetic Study of Apo B100 Containing Lipoprotein Metabolism Using Amino Acids Labeled with Stable Isotopes: Methodological Aspects;Clinical Chemistry and Laboratory Medicine;1998-01-13
2. Method to measure apolipoprotein B-48 and B-100 secretion rates in an individual mouse: evidence for a very rapid turnover of VLDL and preferential removal of B-48- relative to B-100-containing lipoproteins.;Journal of Lipid Research;1996-01
3. Evaluation of apoA-I kinetics in humans using simultaneous endogenous stable isotope and exogenous radiotracer methods.;Journal of Lipid Research;1993-12
4. Measurement of human apolipoprotein B-48 and B-100 kinetics in triglyceride-rich lipoproteins using [5,5,5-2H3]leucine.;Journal of Lipid Research;1992-06
5. Metabolism of apolipoprotein B in members of a family with accelerated atherosclerosis: Influence of apolipoprotein E-3/E-2 pattern;Metabolism;1992-03
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