Evidence of two mechanisms of prostaglandin release in an in vitro model of muscle damage. Possible therapeutic implications-Reference-Cited by-同舟云学术

Evidence of two mechanisms of prostaglandin release in an in vitro model of muscle damage. Possible therapeutic implications

Published:1994-09 Issue:5-6 Volume:4 Page:483-488
ISSN:0960-8966
Container-title:Neuromuscular Disorders
language:en
Short-container-title:Neuromuscular Disorders

Author:

Majumdar Ramanath,Cwik Valerie A.,Brooke Michael H.

Publisher

Elsevier BV

Subject

Genetics(clinical),Clinical Neurology,Neurology,Pediatrics, Perinatology, and Child Health

Reference44 articles.

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4. Protection against ischemic hippocampal CA1 damage in the rat with a new non-NMDA antagonist, NBQX;Diemer;Acta Neurol Scand,1992

5. The effect of the NMDA receptor antagonist MK-801 on cerebral blood flow and infarct volume in experimental focal stroke;Buchan;Brain Res,1992

Cited by 3 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Neurotrophic factors decrease the release of creatine kinase and prostaglandin E2 from metabolically stressed muscle;Neuromuscular Disorders;1998-02

2. Evidence of a temperature-sensitive step in the release of prostaglandin E2 in calcium ionophore-stimulated rat muscle;Prostaglandins, Leukotrienes and Essential Fatty Acids;1995-08

3. Effects of calcium channel antagonists on the release of prostaglandin e2 from metabolically stressed muscle;Biochemical Pharmacology;1994-07