Deficient prolylcarboxypeptidase gene and protein expression in left ventricles of spontaneously hypertensive rats (SHR)-Reference-Cited by-同舟云学术

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Deficient prolylcarboxypeptidase gene and protein expression in left ventricles of spontaneously hypertensive rats (SHR)

Published:2014-11 Issue: Volume:61 Page:69-74
ISSN:0196-9781
Container-title:Peptides
language:en
Short-container-title:Peptides

Author:

Marangoni Rossana Anderson,Santos Rosangela Aparecida,Piccolo Camila

Funder

Conselho Nacional de Desenvolvimento Científico e Tecnológico (CNPq)

Publisher

Elsevier BV

Subject

Cellular and Molecular Neuroscience,Endocrinology,Physiology,Biochemistry

Reference36 articles.

1. Murine prolylcarboxypeptidase depletion induces vascular dysfunction with hypertension and faster arterial thrombosis;Adams;Blood,2011

2. Angiotensin peptides in spontaneously hypertensive and normotensive Donryu rats;Campbell;Hypertension,1995

3. Essential hypertension. Part I: Definition and etiology;Carretero;Circulation,2000

4. Novel mechanism of action of ACE and its inhibitors;Carretero;Am J Physiol Heart Circ Physiol,2005

5. Molecular and cellular mechanisms of myocardial failure;Colucci;Am J Cardiol,1997

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1. Prolylcarboxypeptidase Alleviates Hypertensive Cardiac Remodeling by Regulating Myocardial Tissue Angiotensin II;Journal of the American Heart Association;2023-06-20

2. Role of the renin-angiotensin system in the development of severe COVID-19 in hypertensive patients;American Journal of Physiology-Lung Cellular and Molecular Physiology;2020-10-01

3. Prolylcarboxypeptidase deficiency is associated with increased blood pressure, glomerular lesions, and cardiac dysfunction independent of altered circulating and cardiac angiotensin II;Journal of Molecular Medicine;2017-02-03

4. Antithrombotic potential of the contact activation pathway;Current Opinion in Hematology;2016-09

5. A Cross-sectional Study of KLKB1 and PRCP Polymorphisms in Patient Samples with Cardiovascular Disease;FRONT MED-LAUSANNE;2016

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