Lack of interaction between peripheral injection of CCK and obestatin in the regulation of gastric satiety signaling in rodents-Reference-Cited by-同舟云学术

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Lack of interaction between peripheral injection of CCK and obestatin in the regulation of gastric satiety signaling in rodents

Published:2006-11 Issue:11 Volume:27 Page:2811-2819
ISSN:0196-9781
Container-title:Peptides
language:en
Short-container-title:Peptides

Author:

Gourcerol G.,Million M.,Adelson D.W.,Wang Y.,Wang L.,Rivier J.,St-Pierre D.H.,Taché Y

Publisher

Elsevier BV

Subject

Cellular and Molecular Neuroscience,Endocrinology,Physiology,Biochemistry

Reference36 articles.

1. Coordinated gastric and sphincter motility evoked by intravenous CCK-8 as monitored by ultrasonomicrometry in rats;Adelson;Am J Physiol Gastrointest Liver Physiol,2004

2. H2O2 sensitivity of afferent splanchnic C fiber units in vitro;Adelson;J Neurophysiol,1996

3. Ghrelin is an appetite-stimulatory signal from stomach with structural resemblance to motilin;Asakawa;Gastroenterology,2001

4. Urocortin reduces food intake and gastric emptying in lean and ob/ob obese mice;Asakawa;Gastroenterology,1999

5. Synergistic interaction between leptin and cholecystokinin to reduce short-term food intake in lean mice;Barrachina;Proc Natl Acad Sci USA,1997

Cited by 108 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Diverse and Complementary Effects of Ghrelin and Obestatin;Biomolecules;2022-03-29

2. The effects of intra-stomach obestatin administration on intestinal contractility in neonatal piglets fed milk formula;PLOS ONE;2020-03-23

3. UCN3 suppresses food intake in coordination with CCK and the CCK2R in Siberian sturgeon (Acipenser baerii);Comparative Biochemistry and Physiology Part A: Molecular & Integrative Physiology;2019-08

4. Intraperitoneal injection of nesfatin-1 primarily through the CCK-CCK1R signal pathway affects expression of appetite factors to inhibit the food intake of Siberian sturgeon (Acipenser baerii);Peptides;2018-11

5. Obestatin protects and reverses nonalcoholic fatty liver disease and its associated insulin resistance in rats via inhibition of food intake, enhancing hepatic adiponectin signaling, and blocking ghrelin acylation;Archives of Physiology and Biochemistry;2018-02-10

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