Parkinson’s disease: the thalamic components of the limbic loop are severely impaired by α-synuclein immunopositive inclusion body pathology-Reference-Cited by-同舟云学术

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Parkinson’s disease: the thalamic components of the limbic loop are severely impaired by α-synuclein immunopositive inclusion body pathology

Published:2002-03 Issue:2 Volume:23 Page:245-254
ISSN:0197-4580
Container-title:Neurobiology of Aging
language:en
Short-container-title:Neurobiology of Aging

Author:

Rüb U,Del Tredici K,Schultz C,Ghebremedhin E,de Vos R.A.I,Jansen Steur E,Braak H

Publisher

Elsevier BV

Subject

Geriatrics and Gerontology,Developmental Biology,Neurology (clinical),Aging,General Neuroscience

Reference61 articles.

1. Primate spinothalamic pathways. III. Thalamic terminations of the dorsolateral and ventral spinothalamic pathways;Apkarian;J Comp Neurol,1989

2. Immunoelectronmicroscopic demonstration of NACP/α-synuclein-epitopes on the filamentous component of Lewy bodies in Parkinson’s disease and in dementia with Lewy bodies;Arima;Brain Res,1989

3. Aggregation of α-synuclein in Lewy bodies of sporadic Parkinson’s disease and dementia with Lewy Bodies;Baba;Am J Pathol,1998

4. α-Synuclein accumulates in Lewy bodies in Parkinson’s disease and dementia with Lewy bodies but not in Alzheimer’s disease β-amyloid plaque cores;Bayer;Neurosci Lett,1999

5. The specificity of the nonspecific thalamus;Bentivoglio;Prog Brain Res,1991

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