Activated Alzheimer Disease Platelets Retain More Beta Amyloid Precursor Protein-Reference-Cited by-同舟云学术

Activated Alzheimer Disease Platelets Retain More Beta Amyloid Precursor Protein

Published:1997-03 Issue:2 Volume:18 Page:147-153
ISSN:0197-4580
Container-title:Neurobiology of Aging
language:en
Short-container-title:Neurobiology of Aging

Author:

Davies T.A,Long H.J,Sgro K,Rathbun W.H,McMenamin M.E,Seetoo K,Tibbles H,Billingslea A.M,Fine R.E,Fishman J.B,Levesque C.A,Smith S.J,Wells J.M,Simons E.R

Publisher

Elsevier BV

Subject

Geriatrics and Gerontology,Developmental Biology,Neurology (clinical),Aging,General Neuroscience

Reference27 articles.

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2. The amyloid precursor protein of Alzheimer’s disease is released by human platelets;Bush;J. Biol. Chem.,1990

3. An abnormality of plasma amyloid protein precursor in Alzheimer’s disease;Bush;Ann. Neurol.,1992

4. Dementia. A review emphasizing clinicopathologic correlation and brain-behavior relationships;Chui;Arch. Neurol.,1989

5. Stimulated platelets release amyloid β-protein precursor;Cole;Biochem. Biophys. Res. Commun.,1990

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