Life-long overexpression of S100β in Down’s syndrome: implications for Alzheimer pathogenesis-Reference-Cited by-同舟云学术

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Life-long overexpression of S100β in Down’s syndrome: implications for Alzheimer pathogenesis

Published:1998-09 Issue:5 Volume:19 Page:401-405
ISSN:0197-4580
Container-title:Neurobiology of Aging
language:en
Short-container-title:Neurobiology of Aging

Author:

Griffin W.S.T.,Sheng J.G.,McKenzie J.E.,Royston M.C.,Gentleman S.M.,Brumback R.A.,Cork L.C.,Del Bigio M.R.,Roberts G.W.,Mrak R.E.

Publisher

Elsevier BV

Subject

Geriatrics and Gerontology,Developmental Biology,Clinical Neurology,Ageing,General Neuroscience

Reference22 articles.

1. Gene encoding the β subunit of S100 protein is on chromosome 21;Allore;Science,1988

2. S100β stimulates calcium fluxes in glial and neuronal cells;Barger;J. Biol. Chem.,1992

3. S100 is present in developing chicken neurons and Schwann cells and promotes neuron survival in vivo;Bhattacharyya;J. Neurobiol.,1992

4. Neuropathology of Down syndrome and Alzheimer disease;Cork;Am. J. Med. Gen.,1990

5. Gliosis in human brain;da Cunha;Brain Res,1993

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