Isaacs syndrome: A slow potassium channelopathy caused by autoantibodies?-Reference-Cited by-同舟云学术

Isaacs syndrome: A slow potassium channelopathy caused by autoantibodies?

Published:2018-05 Issue:5 Volume:129 Page:956-958
ISSN:1388-2457
Container-title:Clinical Neurophysiology
language:en
Short-container-title:Clinical Neurophysiology

Author:

Nakazato Tomoko,Tsuji Yukiko,Kanai Kazuaki,Noto Yu-ichi,Hoshino Yasunobu,Yamashiro Kazuo,Yokoyama Kazumasa,Nishioka Kenya,Shimo Yasushi,Watanabe Osamu,Mizuno Toshiki,Hattori Nobutaka

Publisher

Elsevier BV

Subject

Physiology (medical),Neurology (clinical),Neurology,Sensory Systems

Reference5 articles.

1. Isaacs syndrome: a review;Ahmed;Muscle Nerve,2015

2. The collapsin response mediator protein 5 onconeural protein is expressed in Schwann cells under axonal signals and regulates axon-Schwann cell interactions;Camdessanché;J Neuropathol Exp Neurol,2012

3. Immunological associations of acquired neuromyotonia (Isaacs' syndrome). Report of five cases and literature review;Newsom-Davis;Brain,1993

4. Abnormal gating of axonal slow potassium current in cramp-fasciculation syndrome;Shimatani;Clin Neurophysiol,2015

5. Ion channel and striational antibodies define a continuum of autoimmune neuromuscular hyperexcitability;Vernino;Muscle Nerve,2002

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