Reconstructing a disease: What essential features of the retinoic acid receptor fusion oncoproteins generate acute promyelocytic leukemia?
Author:
Publisher
Elsevier BV
Subject
Cancer Research,Cell Biology,Oncology
Reference16 articles.
1. Acute myeloid leukemia fusion proteins deregulate genes involved in stem cell maintenance and DNA repair
2. PML Is a Direct p53 Target that Modulates p53 Effector Functions
3. Methyltransferase Recruitment and DNA Hypermethylation of Target Promoters by an Oncogenic Transcription Factor
4. Retinoic Acid Receptors β and γ Do Not Repress, But Instead Activate Target Gene Transcription in Both the Absence and Presence of Hormone Ligand
5. Impairment of p53 acetylation, stability and function by an oncogenic transcription factor
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1. Acute promyelocytic leukemia with FIP1L1::RARA fusion gene: The clinical utility of transcriptome sequencing and bioinformatic analyses;Frontiers in Oncology;2023-01-26
2. Identification of a novel TNRC18‐RARA fusion in acute promyelocytic leukemia lacking t(15;17)(q24;q12)/ PML‐RARA;Molecular Carcinogenesis;2021-01-11
3. Classic and Variants APLs, as Viewed from a Therapy Response;Cancers;2020-04-14
4. RNF8 is responsible for ATRA resistance in variant acute promyelocytic leukemia with GTF2I/RARA fusion, and inhibition of the ubiquitin–proteasome pathway contributes to the reversion of ATRA resistance;Cancer Cell International;2019-04-04
5. Acute promyelocytic leukemia and variant fusion proteins: PLZF-RARα fusion protein at a glance;Seminars in Oncology;2019-04
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