Loss of p21 Permits Carcinogenesis from Chronically Damaged Liver and Kidney Epithelial Cells despite Unchecked Apoptosis-Reference-Cited by-同舟云学术

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Loss of p21 Permits Carcinogenesis from Chronically Damaged Liver and Kidney Epithelial Cells despite Unchecked Apoptosis

Published:2008-07 Issue:1 Volume:14 Page:59-67
ISSN:1535-6108
Container-title:Cancer Cell
language:en
Short-container-title:Cancer Cell

Author:

Willenbring Holger,Sharma Amar Deep,Vogel Arndt,Lee Andrew Y.,Rothfuss Andreas,Wang Zhongya,Finegold Milton,Grompe Markus

Publisher

Elsevier BV

Subject

Cancer Research,Cell Biology,Oncology

Reference42 articles.

1. Involvement of p21 and p27 in the regulation of CDK activity and cell cycle progression in the regenerating liver;Albrecht;Oncogene,1998

2. Long-term therapy with NTBC and tyrosine-restricted diet in a murine model of hereditary tyrosinemia type I;Al-Dhalimy;Mol. Genet. Metab.,2002

3. Unique gene expression patterns in liver and kidney associated with exposure to chemical toxicants;Bartosiewicz;J. Pharmacol. Exp. Ther.,2001

4. PKD1 induces p21(waf1) and regulation of the cell cycle via direct activation of the JAK-STAT signaling pathway in a process requiring PKD2;Bhunia;Cell,2002

5. Radiation-induced cell cycle arrest compromised by p21 deficiency;Brugarolas;Nature,1995

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