Identification of Sox9-Dependent Acinar-to-Ductal Reprogramming as the Principal Mechanism for Initiation of Pancreatic Ductal Adenocarcinoma-Reference-Cited by-同舟云学术

Identification of Sox9-Dependent Acinar-to-Ductal Reprogramming as the Principal Mechanism for Initiation of Pancreatic Ductal Adenocarcinoma

Published:2012-12 Issue:6 Volume:22 Page:737-750
ISSN:1535-6108
Container-title:Cancer Cell
language:en
Short-container-title:Cancer Cell

Author:

Kopp Janel L.,von Figura Guido,Mayes Erin,Liu Fen-Fen,Dubois Claire L.,Morris John P.,Pan Fong Cheng,Akiyama Haruhiko,Wright Christopher V.E.,Jensen Kristin,Hebrok Matthias,Sander Maike

Publisher

Elsevier BV

Subject

Cancer Research,Cell Biology,Oncology

Reference59 articles.

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3. SOX9cre1, a cis-acting regulatory element located 1.1 Mb upstream of SOX9, mediates its enhancement through the SHH pathway;Bien-Willner;Hum. Mol. Genet.,2007

4. The mutant K-ras oncogene causes pancreatic periductal lymphocytic infiltration and gastric mucous neck cell hyperplasia in transgenic mice;Brembeck;Cancer Res.,2003

5. The Nestin progenitor lineage is the compartment of origin for pancreatic intraepithelial neoplasia;Carrière;Proc. Natl. Acad. Sci. USA,2007

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