JNK inhibitor CC-930 reduces fibrosis in a murine model of Nf1-deficient fracture repair-Reference-Cited by-同舟云学术

JNK inhibitor CC-930 reduces fibrosis in a murine model of Nf1-deficient fracture repair

Published:2018-11-01 Issue:4 Volume:16 Page:350-357
ISSN:1214-021X
Container-title:Journal of Applied Biomedicine
language:
Short-container-title:JAB

Author:

Deo Nikita,El-Hoss Jad,Kolind Mille,Mikulec Kathy,Peacock Lauren,Little David G.,Schindeler Aaron

Funder

National Health and Medical Research Council of Australia

Children’s Tumour Foundation

Celgene Corp

Bone Health Foundation (Australia)

Publisher

University of South Bohemia in Ceske Budejovice

Subject

General Pharmacology, Toxicology and Pharmaceutics,General Agricultural and Biological Sciences,General Biochemistry, Genetics and Molecular Biology,General Medicine,General Neuroscience

Link

http://jab.zsf.jcu.cz/doi/10.1016/j.jab.2018.01.006.pdf

Reference36 articles.

1. Preliminary experience with the combined use of recombinant bone morphogenetic protein and bisphosphonates in the treatment of congenital pseudarthrosis of the tibia

2. Increased c-Jun-NH2-Kinase Signaling in Neurofibromatosis-1 Heterozygous Microglia Drives Microglia Activation and Promotes Optic Glioma Proliferation

3. Signal Transduction by the JNK Group of MAP Kinases

4. Combined MEK Inhibition and BMP2 Treatment Promotes Osteoblast Differentiation and Bone Healing inNf1Osx−/−Mice

5. Deterioration of fracture healing in the mouse model of NF1 long bone dysplasia

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