Two novel mutations of SCN9A (Nav1.7) are associated with partial congenital insensitivity to pain-Reference-Cited by-同舟云学术

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Two novel mutations of SCN9A (Nav1.7) are associated with partial congenital insensitivity to pain

Published:2011-03 Issue:3 Volume:15 Page:223-230
ISSN:1090-3801
Container-title:European Journal of Pain
language:en
Short-container-title:

Author:

Staudl Roland,Pricel Donald D.,Janickel David,Andradel Edgard,Hadjipanayisl Angela G.,Eatonl Will T.,Kaplanl Lee,Wallacel Margaret R.

Publisher

Wiley

Subject

Anesthesiology and Pain Medicine

Reference51 articles.

1. A stop codon mutation in SCN9A causes lack of pain sensation;Ahmad;Hum Mol Genet,2007

2. Afferent pain pathways: a neuroanatomical review;Almeida;Brain Res,2004

3. The role of sodium channels in chronic inflammatory and neuropathic pain;Amir;J Pain,2006

4. An index of empathy for children and adolescents;Bryant;Child Dev,1982

5. The roles of spatial recruitment and discharge frequency in spinal cord coding of pain: a combined electrophysiological and imaging investigation;Coghill;Pain,1993

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1. Novel SCN9A variant associated with congenital insensitivity to pain;Molecular Biology Reports;2023-05-25

2. Novel SCN9A variant associated with congenital insensitivity to pain.;2023-03-01

3. Rare mutations in ATL3, SPTLC2 and SCN9A explaining hereditary sensory neuropathy and congenital insensitivity to pain in a Brazilian cohort;Journal of the Neurological Sciences;2021-08

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