FOXP3 demethylation as a means of identifying quantitative defects in regulatory T cells in acute coronary syndrome-Reference-Cited by-同舟云学术

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FOXP3 demethylation as a means of identifying quantitative defects in regulatory T cells in acute coronary syndrome

Published:2013-07 Issue:1 Volume:229 Page:263-270
ISSN:0021-9150
Container-title:Atherosclerosis
language:en
Short-container-title:Atherosclerosis

Author:

Lü Cai-Xia,Xu Ren-De,Cao Ming,Wang Guan,Yan Feng-Qin,Shang Sha-Sha,Wu Xiao-Fen,Ruan Lei,Quan Xiao-Qing,Zhang Cun-Tai

Funder

National Basic Research Program of China

Natural Science Foundation

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine

Reference30 articles.

1. Global and regional burden of disease and risk factors, 2001: systematic analysis of population health data;Lopez;Lancet,2006

2. Inflammation, atherosclerosis, and coronary artery disease;Hansson;N Engl J Med,2005

3. Effector memory t cells are associated with atherosclerosis in humans and animal models;Ammirati;J Am Heart Assoc,2012

4. Naturally arising CD4+ regulatory t cells for immunologic self-tolerance and negative control of immune responses;Sakaguchi;Annu Rev Immunol,2004

5. Control of regulatory T cell development by the transcription factor Foxp3;Hori;Science,2003

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3. Epigenetic modifications as therapeutic targets in atherosclerosis: a focus on DNA methylation and non-coding RNAs;Frontiers in Cardiovascular Medicine;2023-05-25

4. Downregulation of TIGIT Expression in FOXP3+Regulatory T Cells in Acute Coronary Syndrome;Journal of Inflammation Research;2022-02

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