Copper chelation by tetrathiomolybdate inhibits vascular inflammation and atherosclerotic lesion development in apolipoprotein E-deficient mice-Reference-Cited by-同舟云学术

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Copper chelation by tetrathiomolybdate inhibits vascular inflammation and atherosclerotic lesion development in apolipoprotein E-deficient mice

Published:2012-08 Issue:2 Volume:223 Page:306-313
ISSN:0021-9150
Container-title:Atherosclerosis
language:en
Short-container-title:Atherosclerosis

Author:

Wei Hao,Zhang Wei-Jian,McMillen Timothy S.,LeBoeuf Renee C.,Frei Balz

Funder

National Center for Complementary and Alternative Medicine (NCCAM)

NIH

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine

Reference31 articles.

1. Atherosclerosis;Lusis;Nature,2000

2. The expression of the adhesion molecules ICAM-1, VCAM-1, PECAM, and E-selectin in human atherosclerosis;Davies;J Pathol,1993

3. MCP-1 deficiency reduces susceptibility to atherosclerosis in mice that overexpress human apolipoprotein B;Gosling;J Clin Invest,1999

4. ICAM-1 deficiency reduces atherosclerotic lesions in double-knockout mice (ApoE(−/−)/ICAM-1(−/−)) fed a fat or a chow diet;Bourdillon;Arterioscler Thromb Vasc Biol,2000

5. NF-kappaB: pivotal mediator or innocent bystander in atherogenesis?;Collins;J Clin Invest,2001

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