The role of divalent cations in controlling amphibian lens membrane permeability; The mechanisms of toxic cataracts-Reference-Cited by-同舟云学术

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The role of divalent cations in controlling amphibian lens membrane permeability; The mechanisms of toxic cataracts

Published:1983-04 Issue:4 Volume:36 Page:595-605
ISSN:0014-4835
Container-title:Experimental Eye Research
language:en
Short-container-title:Experimental Eye Research

Author:

Jacob T.J.C.,Duncan G.

Publisher

Elsevier BV

Subject

Cellular and Molecular Neuroscience,Sensory Systems,Ophthalmology

Reference28 articles.

1. Divalent ion currents and the delayed potassium conductance in an Aplysia neurone;Adams;J. Physiol.,1980

2. Intracellular Ca2+ activates a fast voltage-sensitive K+ current in vertebrate sympathetic neurones;Adams;Nature (London),1982

3. Augenveränderungen durch Kobaltchlorid;Alagna;Arch. Ottal.,1956

4. Traumatic cataract;Bellows,1975

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1. Etiopathogenesis of cataract: An appraisal;Indian Journal of Ophthalmology;2014

2. Differentiation-dependent changes in the membrane properties of fiber cells isolated from the rat lens;American Journal of Physiology-Cell Physiology;2008-05

3. Levels of Iron, Zinc, and Copper in Aqueous Humor, Lens, and Serum in Nondiabetic and Diabetic Patients: Their Relation to Cataract;Biological Trace Element Research;2005

4. Thapsigargin inhibits a potassium conductance and stimulates calcium influx in the intact rat lens;The Journal of Physiology;1999-04

5. Pathophysiology of cataracts: Copper ion and peroxidation in diabetics;Japanese Journal of Ophthalmology;1997-06

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