The KIF5B-RET Fusion Gene Mutation as a Novel Mechanism of Acquired EGFR Tyrosine Kinase Inhibitor Resistance in Lung Adenocarcinoma-Reference-Cited by-同舟云学术

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The KIF5B-RET Fusion Gene Mutation as a Novel Mechanism of Acquired EGFR Tyrosine Kinase Inhibitor Resistance in Lung Adenocarcinoma

Published:2019-01 Issue:1 Volume:20 Page:e73-e76
ISSN:1525-7304
Container-title:Clinical Lung Cancer
language:en
Short-container-title:Clinical Lung Cancer

Author:

Zhu You-cai,Wang Wen-xian,Zhang Qu-xia,Xu Chun-wei,Zhuang Wu,Du Kai-qi,Chen Gang,Lv Tang-feng,Song Yong

Funder

Medical Scientific Research Foundation

Technology Bureau of Jiaxing City

Publisher

Elsevier BV

Subject

Cancer Research,Pulmonary and Respiratory Medicine,Oncology

Reference23 articles.

1. Global cancer statistics, 2012;Torre;CA Cancer J Clin,2015

2. Genome-wide association study of genetic predictors of overall survival for non-small cell lung cancer in never smokers;Wu;Cancer Res,2013

3. EGF receptor gene mutations are common in lung cancers from never smokers and are associated with sensitivity of tumors to gefitinib and erlotinib;Pao;Proc Natl Acad Sci U S A,2004

4. A prospective, molecular epidemiology study of EGFR mutations in Asian patients with advanced non-small-cell lung cancer of adenocarcinoma histology (PIONEER);Shi;J Thorac Oncol,2014

5. Gefitinib or carboplatin paclitaxel in pulmonary adenocarcinoma;Mok;N Engl J Med,2009

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4. HER2-positive breast cancer progresses rapidly after pyrotinib resistance: acquired RET gene fusion and TP53 gene mutation are potential reasons;Anti-Cancer Drugs;2023-01-24

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