TCDD promotes liver fibrosis through disordering systemic and hepatic iron homeostasis-Reference-Cited by-同舟云学术

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TCDD promotes liver fibrosis through disordering systemic and hepatic iron homeostasis

Published:2020-08 Issue: Volume:395 Page:122588
ISSN:0304-3894
Container-title:Journal of Hazardous Materials
language:en
Short-container-title:Journal of Hazardous Materials

Author:

Li Changying,Liu Yingying,Dong Zheng,Xu Ming,Gao Ming,Cong Min,Liu Sijin

Funder

National Natural Science Foundation of China

Beijing Natural Science Foundation

Chinese Academy of Sciences

Publisher

Elsevier BV

Subject

Health, Toxicology and Mutagenesis,Pollution,Waste Management and Disposal,Environmental Chemistry,Environmental Engineering

Reference54 articles.

1. TCDD-elicited effects on liver, serum, and adipose lipid composition in C57BL/6 mice;Angrish;Toxicol. Sci.,2013

2. 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) or diethylstilbestrol (DES) cause similar hematopoietic hypocellularity and hepatocellular changes in murine fetal liver, but differentially affect gene expression;Besteman;Toxicol. Pathol.,2007

3. Non-transferrin bound iron: a key role in iron overload and iron toxicity;Brissot;Biochim. Biophys. Acta,2012

4. Embryotoxicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD): the embryonic vasculature is a physiological target for TCDD-induced DNA damage and apoptotic cell death in Medaka (Orizias latipes);Cantrell;Toxicol. Appl. Pharmacol.,1996

5. Resveratrol inhibits TCDD-induced expression of CYP1A1 and CYP1B1 and catechol estrogen-mediated oxidative DNA damage in cultured human mammary epithelial cells;Chen;Carcinogenesis,2004

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