Vagaries of Vav
Author:
Publisher
Elsevier BV
Subject
General Agricultural and Biological Sciences,General Biochemistry, Genetics and Molecular Biology
Reference20 articles.
1. SH2 and SH3 domains: elements that control interactions of cytoplasmic signaling proteins;Koch;Science,1991
2. Oncogenes and signal transduction;Cantley;Cell,1991
3. BCR sequences essential for transformation by the BCR-ABL oncogene bind to the ABL SH2 regulatory domain in a nonphosphotyrosine-dependent manner;Pendergast;Cell,1991
4. vav, a novel human oncogene derived from a locus ubiquitously expressed in hematopoietic cells;Katzav;EMBO J,1989
5. Mechanism of activation of the vav protooncogene;Coppola;Cell Growth Diff,1991
Cited by 17 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. A targeted κ immunoglobulin gene containing a deletion of the nuclear matrix association region exhibits spontaneous hyper-recombination in pre-B cells;Molecular Immunology;1998-07
2. Defects in actin-cap formation in Vav-deficient mice implicate an actin requirement for lymphocyte signal transduction;Current Biology;1998-05
3. Role of GTP-Binding Proteins in FcεRI Signaling;IgE Receptor (FcεRI) Function in Mast Cells and Basophils;1997
4. Vav: Function and Regulation in Hematopoietic Cell Signaling;STEM CELLS;1996-05
5. Enhancement of Protein Kinase C-dependent O2⋅¯ Production in Epstein-Barr Virus-transformed B Lymphocytes by p120Ras-GAP Antisense Oligonucleotide;Journal of Biological Chemistry;1996-04
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