Transformation to “high grade” neuroendocrine carcinoma as an acquired drug resistance mechanism in EGFR-mutant lung adenocarcinoma-Reference-Cited by-同舟云学术

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Transformation to “high grade” neuroendocrine carcinoma as an acquired drug resistance mechanism in EGFR-mutant lung adenocarcinoma

Published:2013-04 Issue:1 Volume:80 Page:1-4
ISSN:0169-5002
Container-title:Lung Cancer
language:en
Short-container-title:Lung Cancer

Author:

Popat S.,Wotherspoon A.,Nutting C.M.,Gonzalez D.,Nicholson A.G.,O’Brien M.

Funder

Royal Marsden Hospital/Institute of Cancer Research NIHR Biomedical Research Centre

Publisher

Elsevier BV

Subject

Cancer Research,Pulmonary and Respiratory Medicine,Oncology

Reference4 articles.

1. Genotypic and histological evolution of lung cancers acquiring resistance to EGFR inhibitors;Sequist;Sci Transl Med,2011

2. EGFR mutations in small-cell lung cancers in patients who have never smoked;Zakowski;N Engl J Med,2006

3. Sequential occurrence of non-small cell and small cell lung cancer with the same EGFR mutation;Morinaga;Lung Cancer,2007

4. International association for the study of lung cancer/american thoracic society/european respiratory society international multidisciplinary classification of lung adenocarcinoma;Travis;J Thorac Oncol,2011

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3. Clonality Analysis for the Relationship between the Pulmonary Combined Neuroendocrine Carcinoma and “the So-Called Reported Histologic Transformation”;Cancers;2023-11-29

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