Acquired KRAS mutation and loss of low-level MET amplification after durable response to crizotinib in a patient with lung adenocarcinoma
Author:
Funder
Pfizer
Bristol-Myers Squibb
Novartis
MSD
Janssen Pharmaceuticals
Publisher
Elsevier BV
Subject
Cancer Research,Pulmonary and Respiratory Medicine,Oncology
Reference13 articles.
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2. Targeting MET in lung cancer: will expectations finally be MET?;Drilon;J. Thorac. Oncol.,2017
3. Phase (Ph) I study of the safety and efficacy of the cMET inhibitor capmatinib (INC280) in patients (pts) with advanced cMET+ non-small cell lung cancer (NSCLC);Schuler;J. Clin. Oncol.,2016
4. Efficacy and safety of crizotinib in patients with advanced c-MET-amplified non-small cell lung cancer (NSCLC);Camidge;J. Clin. Oncol.,2014
5. MET amplification status in therapy-naive adeno- and squamous cell carcinomas of the lung;Schildhaus;Clin. Cancer Res.,2015
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1. Resistance to MET inhibition in MET-dependent NSCLC and therapeutic activity after switching from type I to type II MET inhibitors;European Journal of Cancer;2023-01
2. KRAS Inhibitor Resistance in MET-Amplified KRASG12C Non–Small Cell Lung Cancer Induced By RAS- and Non–RAS-Mediated Cell Signaling Mechanisms;Clinical Cancer Research;2021-08-07
3. Targeting Infrequent Driver Alterations in Non-Small Cell Lung Cancer;Trends in Cancer;2021-05
4. Crizotinib;Reactions Weekly;2019-06
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