HOTAIR induces EGFR-TKIs resistance in non-small cell lung cancer through epithelial-mesenchymal transition-Reference-Cited by-同舟云学术

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HOTAIR induces EGFR-TKIs resistance in non-small cell lung cancer through epithelial-mesenchymal transition

Published:2020-09 Issue: Volume:147 Page:99-105
ISSN:0169-5002
Container-title:Lung Cancer
language:en
Short-container-title:Lung Cancer

Author:

Wang Qi,Li Xuefei,Ren Shengxiang,Su Chunxia,Li Chunyu,Li Wei,Yu Jia,Cheng Ningning,Zhou Caicun

Funder

Shanghai Anticancer Association EYAS PROJECT

“Dream Tutor” New Person Cultivation Program of Shanghai Pulmonary Hospital

National Natural Science Foundation of China

Natural Science Foundation of Tianjin

Shanghai Municipal Commission of Health and Family Planning

Shanghai Committee of Science and Technology

Publisher

Elsevier BV

Subject

Cancer Research,Pulmonary and Respiratory Medicine,Oncology

Reference38 articles.

1. Erlotinib versus chemotherapy as first-line treatment for patients with advanced EGFR mutation-positive non-small-cell lung cancer (OPTIMAL, CTONG-0802): a multicentre, open-label, randomised, phase 3 study;Zhou;Lancet Oncol.,2011

2. American Society of Clinical Oncology provisional clinical opinion: epidermal growth factor receptor (EGFR) Mutation testing for patients with advanced non-small-cell lung cancer considering first-line EGFR tyrosine kinase inhibitor therapy;Keedy;J. Clin. Oncol.,2011

3. Relative abundance of EGFR mutations predicts benefit from gefitinib treatment for advanced non-small-cell lung cancer;Zhou;J. Clin. Oncol.,2011

4. Clinical responses to EGFR-tyrosine kinase inhibitor retreatment in non-small cell lung cancer patients who benefited from prior effective gefitinib therapy: a retrospective analysis;Watanabe;BMC Cancer,2011

5. Resistance to epidermal growth factor receptor tyrosine kinase inhibitors in non-small cell lung cancer;Hammerman;Clin. Cancer Res.,2009

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