Crizotinib inhibits activation of MET pathway caused by MET extracellular SEMA domain duplication-Reference-Cited by-同舟云学术

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Crizotinib inhibits activation of MET pathway caused by MET extracellular SEMA domain duplication

Published:2020-09 Issue: Volume:147 Page:64-70
ISSN:0169-5002
Container-title:Lung Cancer
language:en
Short-container-title:Lung Cancer

Author:

Lin Jiatong,Lyu Yingcheng,Li Lin,Wu Duoguang,Chen Jiewen,Lin Tengjiao,Huang Zhan,Hu Jinwei,Wang Wenjing,Lin Huayue,Wang Minghui

Funder

National Natural Science Foundation of China

Natural Science Foundation of Guangdong Province

Guangdong Science and Technology Department

Guangdong Basic and Applied Basic Research Foundation

Fundamental Research Funds for the Central Universities

Publisher

Elsevier BV

Subject

Cancer Research,Pulmonary and Respiratory Medicine,Oncology

Reference31 articles.

1. Somatic mutations lead to an oncogenic deletion of met in lung cancer;Kong-Beltran;Cancer Res.,2006

2. MET amplification identifies a small and aggressive subgroup of esophagogastric adenocarcinoma with evidence of responsiveness to crizotinib;Lennerz;J. Clin. Oncol.,2011

3. Impact of MET amplification on gastric cancer: possible roles as a novel prognostic marker and a potential therapeutic target;Lee;Oncol. Rep.,2011

4. MET amplification leads to gefitinib resistance in lung cancer by activating ERBB3 signaling;Engelman;Science,2007

5. Amplification of the MET receptor drives resistance to anti-EGFR therapies in colorectal cancer;Bardelli;Cancer Discov.,2013

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