Secondary atypical hemolytic uremic syndromes in the era of complement blockade
Author:
Publisher
Elsevier BV
Subject
Nephrology
Reference9 articles.
1. Atypical hemolytic uremic syndrome and C3 glomerulopathy: conclusions from a Kidney Disease: Improving Global Outcomes (KDIGO) Controversies Conference;Goodship;Kidney Int,2017
2. Thrombotic microangiopathy and the kidney;Brocklebank;Clin J Am Soc Nephrol,2018
3. Haemolytic uraemic syndrome;Fakhouri;Lancet,2017
4. Atypical and secondary hemolytic uremic syndromes have a distinct presentation and no common genetic risk factors;Le Clech;Kidney Int,2019
5. Predisposition to atypical hemolytic uremic syndrome involves the concurrence of different susceptibility alleles in the regulators of complement activation gene cluster in 1q32;Esparza-Gordillo;Hum Mol Genet,2005
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1. Rational use of eculizumab in secondary atypical hemolytic uremic syndrome;Frontiers in Immunology;2024-01-11
2. Endothelial activation and damage as a common pathological substrate in different pathologies and cell therapy complications;Frontiers in Medicine;2023-11-14
3. The Rationale of Complement Blockade of the MCPggaac Haplotype following Atypical Hemolytic Uremic Syndrome of Three Southeastern European Countries with a Literature Review;International Journal of Molecular Sciences;2023-08-22
4. Complement‐driven hemolytic uremic syndrome;American Journal of Hematology;2023-03-31
5. The loss of glycocalyx integrity impairs complement factor H binding and contributes to cyclosporine-induced endothelial cell injury;Frontiers in Medicine;2023-02-13
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