Inhibition of transforming growth factor β1 signaling in resident interstitial cells attenuates profibrotic gene expression and preserves erythropoietin production during experimental kidney fibrosis in mice
Author:
Funder
Deutsche Forschungsgemeinschaft
Publisher
Elsevier BV
Subject
Nephrology
Reference68 articles.
1. Mechanisms of fibrosis: the role of the pericyte;Schrimpf;Curr Opin Nephrol Hypertens,2011
2. Renal fibrosis is not reduced by blocking transforming growth factor-β signaling in matrix-producing interstitial cells;Neelisetty;Kidney Int,2015
3. Extracellular matrix in kidney fibrosis: more than just a scaffold;Bülow;J Histochem Cytochem,2019
4. Mechanisms of renal fibrosis;Humphreys;Annu Rev Physiol,2018
5. The interstitial response to renal injury: fibroblast–like cells show phenotypic changes and have reduced potential for erythropoietin gene expression;Maxwell;Kidney Int,1997
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