Deficiency of the kidney tubular angiotensin II type1 receptor–associated protein ATRAP exacerbates streptozotocin-induced diabetic glomerular injury via reducing protective macrophage polarization

Author:

Haruhara Kotaro,Suzuki Toru,Wakui Hiromichi,Azushima Kengo,Kurotaki Daisuke,Kawase Wataru,Uneda Kazushi,Kobayashi Ryu,Ohki Kohji,Kinguchi Sho,Yamaji Takahiro,Kato Ikuma,Ohashi Kenichi,Yamashita Akio,Tamura Tomohiko,Tsuboi NobuoORCID,Yokoo Takashi,Tamura Kouichi

Publisher

Elsevier BV

Subject

Nephrology

Reference72 articles.

1. The protective effects of beta-casomorphin-7 against glucose-induced renal oxidative stress in vivo and vitro;Zhang;PLoS One,2013

2. High glucose augments angiotensinogen in human renal proximal tubular cells through hepatocyte nuclear factor-5;Wang;PLoS One,2017

3. High glucose upregulates upstream stimulatory factor 2 in human renal proximal tubular cells through angiotensin II-dependent activation of CREB;Visavadiya;Nephron Exp Nephrol,2011

4. High glucose instigates tubulointerstitial injury by stimulating hetero-dimerization of adiponectin and angiotensin II receptors;Zha;Biochem Biophys Res Commun,2017

5. The Collaborative Study Group. The effect of angiotensin-converting-enzyme inhibition on diabetic nephropathy;Lewis;N Engl J Med,1993

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