Myeloid expression of the anti-apoptotic protein Mcl1 is required in anti-myeloperoxidase vasculitis but myeloperoxidase inhibition is not protective-Reference-Cited by-同舟云学术

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Myeloid expression of the anti-apoptotic protein Mcl1 is required in anti-myeloperoxidase vasculitis but myeloperoxidase inhibition is not protective

Published:2023-01 Issue:1 Volume:103 Page:134-143
ISSN:0085-2538
Container-title:Kidney International
language:en
Short-container-title:Kidney International

Author:

Florez-Barros Fernanda,Bearder Siobhan,Kull Bengt,Freeman Adrian,Mócsai Attila,Robson Michael G.

Funder

British Heart Foundation

NIHR Biomedical Research Centre, Royal Marsden NHS Foundation Trust/Institute of Cancer Research

King's College London

Medical Research Council

Guy's and St Thomas' NHS Foundation Trust

National Institute for Health and Care Research

Publisher

Elsevier BV

Subject

Nephrology

Reference21 articles.

1. Anti-neutrophil cytoplasmic autoantibodies induce neutrophils to degranulate and produce oxygen radicals in vitro;Falk;Proc Natl Acad Sci U S A,1990

2. Antineutrophil cytoplasmic autoantibodies specific for myeloperoxidase cause glomerulonephritis and vasculitis in mice;Xiao;J Clin Invest,2002

3. How anti-neutrophil cytoplasmic autoantibodies activate neutrophils;Kettritz;Clin Exp Immunol,2012

4. Neutrophils are not consistently activated by antineutrophil cytoplasmic antibodies in vitro;Popat;Ann Rheum Dis,2019

5. The role of neutrophils in the induction of glomerulonephritis by anti-myeloperoxidase antibodies;Xiao;Am J Pathol,2005

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1. The conclusion that myeloperoxidase inhibition is not protective against anti-neutrophil cytoplasmic autoantibody–associated vasculitis is premature;Kidney International;2023-10

2. The authors reply;Kidney International;2023-10

3. A Novel Spatial Search Algorithm for Computing Potentially Transcribed Gene Combinations: A Molecular Biology Network for Behcet's Disease as an Example;SSRN Electronic Journal;2023

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