Notch signaling in bone marrow–derived FSP-1 cells initiates neointima formation in arteriovenous fistulas-Reference-Cited by-同舟云学术

Notch signaling in bone marrow–derived FSP-1 cells initiates neointima formation in arteriovenous fistulas

Published:2019-06 Issue:6 Volume:95 Page:1347-1358
ISSN:0085-2538
Container-title:Kidney International
language:en
Short-container-title:Kidney International

Author:

Liang Ming,Guo Qunying,Huang Fengzhang,Han Guofeng,Song Ke,Luo Jinlong,Cheng Hunter,Hu Hongzhen,Peden Eric K.,Chen Changyi,Mitch William E.,Du Jie,Fu Xiaodong,Truong Luan,Cheng Jizhong

Funder

American Heart Association

National Institutes of Health

National Natural Science Foundation of China

Natural Science Foundation of Guangdong Province

Publisher

Elsevier BV

Subject

Nephrology

Reference39 articles.

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2. Increasing arteriovenous fistulas in hemodialysis patients: problems and solutions;Allon;Kidney Int,2002

3. Chronic kidney disease accelerates endothelial barrier dysfunction in a mouse model of an arteriovenous fistula;Liang;Am J Physiol Renal Physiol,2013

4. CKD accelerates development of neointimal hyperplasia in arteriovenous fistulas;Kokubo;J Am Soc Nephrol,2009

5. Migration of smooth muscle cells from the arterial anastomosis of arteriovenous fistulas requires Notch activation to form neointima;Liang;Kidney Int,2015

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