Neutrophils in cisplatin AKI—mediator or marker?
Author:
Publisher
Elsevier BV
Subject
Nephrology
Reference9 articles.
1. Dendritic cell protection from cisplatin nephrotoxicity is independent of neutrophils;Tadagavadi;Toxins,2015
2. Innate IL-17a-producing leukocytes promote acute kidney injury via inflammasome and Toll-like receptor activation;Chan;Am J Pathol,2014
3. Cisplatin-induced acute renal failure is associated with an increase in the cytokines interleukin (IL)-1beta, IL-18, IL-6, and neutrophil infiltration in the kidney;Faubel;J Pharmacol Exp Ther,2007
4. Dexamethasone ameliorates renal ischemia-reperfusion injury;Kumar;J Am Soc Nephrol,2009
5. The leukotriene B4—leukotriene B4 receptor axis promotes cisplatin-induced acute kidney injury by modulating neutrophil recruitment;Deng;Kidney Int,2017
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