Fraxetin reduces endometriotic lesions through activation of ER stress, induction of mitochondria-mediated apoptosis, and generation of ROS-Reference-Cited by-同舟云学术

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Fraxetin reduces endometriotic lesions through activation of ER stress, induction of mitochondria-mediated apoptosis, and generation of ROS

Published:2024-01 Issue: Volume:123 Page:155187
ISSN:0944-7113
Container-title:Phytomedicine
language:en
Short-container-title:Phytomedicine

Author:

Ham Jiyeon^ORCID,Park Wonhyoung,Song Jisoo,Kim Hee Seung,Song Gwonhwa,Lim Whasun^ORCID,Park Soo Jin^ORCID,Park Sunwoo

Funder

Ministry of Oceans and Fisheries

Ministry of Science, ICT and Future Planning

Ministry of Health and Welfare

Korea Health Industry Development Institute

National Research Foundation of Korea

Ministry of Education

Korea Institute of Marine Science and Technology promotion

Publisher

Elsevier BV

Subject

Complementary and alternative medicine,Drug Discovery,Pharmaceutical Science,Pharmacology,Molecular Medicine

Reference47 articles.

1. New understanding of diagnosis, treatment and prevention of endometriosis;Amro;Int. J. Environ. Res. Public Health,2022

2. Role of AMPK/mTOR, mitochondria, and ROS in the pathogenesis of endometriosis;Assaf;Life Sci.,2022

3. Inhibition of PI3K/AKT/mTOR pathway for the treatment of endometriosis;Barra;Br. J. Pharmacol.,2018

4. The role of mitogen-activated protein kinase signaling pathway in endometriosis;Bora;J. Obstet. Gynaecol. Res.,2021

5. Pathogenesis and pathophysiology of endometriosis;Burney;Fertil. Steril.,2012

Cited by 3 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Bioinformatic analysis reveals endoplasmic reticulum stress-related molecular cluster and immune characterization in endometriosis：implications for disease subtyping and therapeutic strategies;2024-04-12

2. Autophagy regulation and redox perturbation by transcrocetin suppress the growth of endometriosis;Biomedicine & Pharmacotherapy;2024-04

3. Health benefits of fraxetin: From chemistry to medicine;Archiv der Pharmazie;2024-03-19

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