Ginsenoside Rg3 antagonizes adriamycin-induced cardiotoxicity by improving endothelial dysfunction from oxidative stress via upregulating the Nrf2-ARE pathway through the activation of akt-Reference-Cited by-同舟云学术

Ginsenoside Rg3 antagonizes adriamycin-induced cardiotoxicity by improving endothelial dysfunction from oxidative stress via upregulating the Nrf2-ARE pathway through the activation of akt

Published:2015-09 Issue:10 Volume:22 Page:875-884
ISSN:0944-7113
Container-title:Phytomedicine
language:en
Short-container-title:Phytomedicine

Author:

Wang Xiaoying,Chen Lili,Wang Ting,Jiang Xiaoqing,Zhang Han,Li Pan,Lv Bin,Gao Xiumei

Funder

NSFC

Ministry of Education of PRC

Publisher

Elsevier BV

Subject

Complementary and alternative medicine,Drug Discovery,Pharmaceutical Science,Pharmacology,Molecular Medicine

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3. Akt activation by growth factors is a multiple-step process: the role of the PH domain;Bellacosa;Oncogene.,1998

4. Annexin V imaging of acute doxorubicin cardiotoxicity (apoptosis) in rats;Bennink;J. Nucl. Med.,2004

5. Transcription factor Nrf2 coordinately regulates a group of oxidative stress-inducible genes in macrophages;Ishii;J. Biol. Chem.,2000

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