Loss of Sip1 leads to migration defects and retention of ectodermal markers during lens development-Reference-Cited by-同舟云学术

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Loss of Sip1 leads to migration defects and retention of ectodermal markers during lens development

Published:2014-02 Issue: Volume:131 Page:86-110
ISSN:0925-4773
Container-title:Mechanisms of Development
language:en
Short-container-title:Mechanisms of Development

Author:

Manthey Abby L.,Lachke Salil A.,FitzGerald Paul G.,Mason Robert W.,Scheiblin David A.,McDonald John H.,Duncan Melinda K.

Funder

NIH National Center for Research Resources

NIH National Institute of General Medical Sciences

National Science Foundation EPSCoR

NEI Grant

University of Delaware Chemistry-Biology Interface (CBI) Program

Vision Research Core Grant

COBRE Core Grant

Nemours proteomics core

Publisher

Elsevier BV

Subject

Developmental Biology,Embryology

Reference79 articles.

1. Clinical features and management issues in Mowat–Wilson syndrome;Adam;Am. J. Med. Genet. A,2006

2. Ophthalmologic abnormalities in Mowat–Wilson syndrome and a mutation in ZEB2;Ariss;Ophthalmic Genet.,2012

3. Pax6 activity in the lens primordium is required for lens formation and for correct placement of a single retina in the eye;Ashery-Padan;Genes Dev.,2000

4. Differential expression in SAGE: accounting for normal between-library variation;Baggerly;Bioinformatics,2003

5. Pleiotropic and diverse expression of ZFHX1B gene transcripts during mouse and human development supports the various clinical manifestations of the “Mowat–Wilson” syndrome;Bassez;Neurobiol. Dis.,2004

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